Helicobacter pylori cholesteryl glucosides interfere with host membrane phase and affect type IV secretion system function during infection in AGS cells
Author:
Publisher
Wiley
Subject
Molecular Biology,Microbiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2958.2011.07910.x/fullpdf
Reference54 articles.
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3. Functional analysis of the cag pathogenicity island in Helicobacter pylori isolates from patients with gastritis, peptic ulcer, and gastric cancer;Backert;Infect Immun,2004
4. NF-kappaB activation and potentiation of proinflammatory responses by the Helicobacter pylori CagA protein;Brandt;Proc Natl Acad Sci USA,2005
5. cag, a pathogenicity island of Helicobacter pylori, encodes type I-specific and disease-associated virulence factors;Censini;Proc Natl Acad Sci USA,1996
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