Longitudinal effects of hepatitis C virus treatment on hepatic mitochondrial dysfunction assessed by13C-methionine breath test
Author:
Publisher
Wiley
Subject
Pharmacology (medical),Gastroenterology,Hepatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2036.2008.03745.x/fullpdf
Reference21 articles.
1. Epidemiology of hepatitis B and hepatitis B virus infection in United States children;Shepard;Pediatr Infect Dis J,2005
2. Mitochondrial injury, oxidative stress, and antioxidant gene expression are induced by hepatitis C virus core protein;Okuda;Gastroenterology,2002
3. Hepatitis C virus core protein inhibits mitochondrial electron transport and increases reactive oxygen species (ROS) production;Korenaga;J Biol Chem,2005
4. Hepatitis C virus protein expression causes calcium-mediated mitochondrial bioenergetic dysfunction and nitro-oxidative stress;Piccoli;Hepatology,2007
5. Hepatitis C virus core protein increases mitochondrial ROS production by stimulation of Ca2+ uniporter activity;Li;FASEB J,2007
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2. European guideline on indications, performance and clinical impact of 13 C‐breath tests in adult and pediatric patients: An EAGEN, ESNM, and ESPGHAN consensus, supported by EPC;United European Gastroenterology Journal;2021-06
3. Exploring Liver Mitochondrial Function by 13C-Stable Isotope Breath Tests: Implications in Clinical Biochemistry;Methods in Molecular Biology;2021
4. Molecular changes in hepatic metabolism and transport in cirrhosis and their functional importance;World Journal of Gastroenterology;2016
5. Exploring Liver Mitochondrial Function by 13C-Stable Isotope Breath Tests: Implications in Clinical Biochemistry;Methods in Molecular Biology;2014-09-22
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