Elevated plasma 19-hydroxyandrostenedione levels in Cushing's disease: stimulation with ACTH and inhibition with metyrapone
Author:
Publisher
Wiley
Subject
Endocrinology, Diabetes and Metabolism,Endocrinology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2265.1993.tb01005.x/fullpdf
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3. A Reevaluation of the Mineralocorticoid and Hypertensinogenic Potential of 19-Hydroxyandrostenedione*
4. DISSOCIATION BETWEEN PLASMA ADRENAL ANDROGENS AND CORTISOL IN CUSHING'S DISEASE AND ECTOPIC ACTH-PRODUCING TUMOUR: RELATION TO ADRENARCHE
5. Are the Hypokalaemia and Hypertension in Cushing's Disease Caused by Apparent Mineralocorticoid Excess?
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1. 19-hydroxy Steroids in the Aromatase Reaction: Review on Expression and Potential Functions;Journal of the Endocrine Society;2021-03-23
2. Variant of Pre-Clinical Cushing's Syndrome: Hypertension and Hypokalemia Associated with Normoreninemic Normoaldosteronism.;Hypertension Research;2002
3. Evidence for persistent dysfunction of wild-type aldosterone synthase gene in glucocorticoid-treated familial hyperaldosteronism type I;Journal of Hypertension;1997-12
4. Cushing’s Syndrome and Hypertension;Endocrinology and Metabolism Clinics of North America;1994-06
5. Reduced response to metoclopramide and anomalous rising response to upright posture of plasma aldosterone concentration in Japanese patients with aldosterone-producing adenoma;Journal of Clinical Endocrinology & Metabolism;1993-10-01
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