Defective in vivo expression and apparently normal in vitro expression of a newly identified 105-kDa lower lamina lucida protein in dystrophic epidermolysis bullosa
Author:
Publisher
Wiley
Subject
Dermatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2133.1995.tb00717.x/fullpdf
Reference23 articles.
1. Revised clinical and laboratory criteria for subtypes of inherited epidermolysis bullosa: a consensus report by the subcommittee on diagnosis and classification of the National Epidermolysis Bullosa Registry;Fine;J Am Acad Dermatol,1991
2. Immunofluorescence mapping of antigenic determinants within the dermal-epidermal junction in mechanobullous diseases;Hintner;J Invest Dermatol,1981
3. Point mutations in human keratin 14 genes of epidermolysis bullosa simplex patients: genetic and functional analyses;Coulombe;Cell,1991
4. Epidermolysis bullosa simplex: evidence in two families for keratin gene abnormalities;Bonifas;Science,1991
5. 19-DEJ-1, a hemidesmosomeanchoring filament complex-associated monoclonal antibody: definition of a new skin basement membrane antigenic defect in junctional and dystrophic epidermolysis bullosa;Fine;Arch Dermatol,1989
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Fibroblast-Based Cell Therapy Strategy for Recessive Dystrophic Epidermolysis Bullosa;Dermatologic Clinics;2010-04
2. Acquired skin disease of hemidesmosomes;Journal of Dermatological Science;1999-06
3. Hereditary skin diseases of hemidesmosomes;Journal of Dermatological Science;1999-06
4. The 105-kDa Basement Membrane Autoantigen p105 Is N-Terminally Homologous to a Tumor-Associated Antigen;Journal of Investigative Dermatology;1996-08
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