Essential lysine residues within transmembrane helix 1 of diphtheria toxin facilitate COPI binding and catalytic domain entry
Author:
Publisher
Wiley
Subject
Molecular Biology,Microbiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2958.2010.07159.x/fullpdf
Reference31 articles.
1. Interleukin 2 receptor-targeted cytotoxicity. Interleukin 2 receptor-mediated action of a diphtheria toxin-related interleukin 2 fusion protein;Bacha;J Exp Med,1988
2. Coatomer, the coat protein of COPI transport vesicles, discriminates endoplasmic reticulum residents from p24 proteins;Béthune;Mol Cell Biol,2006a
3. COPI-mediated transport;Béthune;J Membr Biol,2006b
4. Coatomer interaction with di-lysine endoplasmic reticulum retention motifs;Cosson;Science,1994
5. ADP-ribosylation factor, a small GTP-binding protein, is required for binding of coatomer protein beta-COP to Golgi membranes;Donaldson;Proc Natl Acad Sci USA,1992
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