Marrow Cells from Patients with Untreated Pernicious Anaemia Cannot Use Tetrahydrofolate Normally
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2141.1980.tb06008.x/fullpdf
Reference22 articles.
1. MEGALOBLASTIC HÆMOPOIESIS IN PATIENTS RECEIVING NITROUS OXIDE
2. Reactions of gases in solution. Part III. Some reactions of nitrous oxide with transition-metal complexes
3. SELECTIVE INACTIVATION OF VITAMIN B12 IN RATS BY NITROUS OXIDE
4. Inactivation of Methionine Synthase by Nitrous Oxide
5. Impaired deoxyuridine utilization in the B12-inactivated rat and its correction by folate analogues
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1. Deoxyuridine suppression test on isolated rat bone marrow cells and the in vitro effect of bidisomide;Toxicology in Vitro;1994-04
2. FOLINIC ACID PROTECTION AGAINST HEMATOPOIETIC DEPRESSION INDUCED BY NITROUS-OXIDE IN RATS;ANESTH ANALG;1993
3. Correction of the DNA synthesis defect in vitamin B12deficiency by tetrahydrofolate: evidence in favour of the methyl-folate trap hypothesis as the cause of megaloblastic anaemia in vitamin B12deficiency;British Journal of Haematology;1993-04
4. Iron Deficiency and Megaloblastic Anemias;Biochemical Pharmacology of Blood and Bloodforming Organs;1992
5. In vitro DNA synthesis by megaloblastic bone marrow: Effect of folates and cobalamins on thymidine incorporation and de novo thymidylate synthesis;American Journal of Hematology;1989-05
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