The Neurotoxicant, Cuprizone, as a Model to Study Demyelination and Remyelination in the Central Nervous System
Author:
Publisher
Wiley
Subject
Clinical Neurology,Pathology and Forensic Medicine,General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1750-3639.2001.tb00385.x/fullpdf
Reference45 articles.
1. Blood-brain barrier permeability during cuprizone-induced demyelination. Implications for the pathogenesis of immune-mediated demyelinating diseases;Bakker;J Neurol Sci,1987
2. Observations on oligodendrocyte degeneration, the resolution of status spongiosus and remyelination in cuprizone intoxication in mice;Blakemore;J Neurocytol,1972
3. Remyelination of the superior cerebellar peduncle in the mouse following demyelination induced by feeding cuprizone;Blakemore;J Neurol Sci,1973a
4. Demyelination of the superior cerebellar peduncle in the mouse induced by cuprizone;Blakemore;J Neurol Sci,1973b
5. Remyelination of the suprerior cerebellar peduncle in old mice following demyelination induced by cuprizone;Blakemore;J Neurol Sci,1974
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