CD40L is Critical for Protection from Demyelinating Disease and Development of Spontaneous Remyelination in a Mouse Model of Multiple Sclerosis-Reference-Cited by-同舟云学术

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CD40L is Critical for Protection from Demyelinating Disease and Development of Spontaneous Remyelination in a Mouse Model of Multiple Sclerosis

Published:2006-04-05 Issue:1 Volume:10 Page:1-15
ISSN:1015-6305
Container-title:Brain Pathology
language:en
Short-container-title:

Author:

Drescher Kristen M.,Zoecklein Laurie J.,Pavelko Kevin D.,Rivera-Quinones Cynthia,Hollenbaugh Diane,Rodriguez Moses

Publisher

Wiley

Subject

Neurology (clinical),Pathology and Forensic Medicine,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1750-3639.2000.tb00238.x/fullpdf

Reference61 articles.

1. Targeting of IgMk antibodies to oligodendrocytes promotes central nervous system remyelination;Asakura;J Neurosci,1998

2. Identification of Theiler's virus infected cells in the central nervous system of the mouse during demyelinating disease;Aubert;Microb Pathog,1987

3. FVB mice transgenic for the H-2Db gene become resistant to persistent infection by Theiler's virus;Azoulay;J Virol,1994

4. CD40 ligand is required for protective cell-mediated immunity to Leishmania major;Campbell;Immunity,1996

5. Characterization of B lymphocytes present in the demyelinating lesionS induced by Theiler's virus;Cash;J Immunol,1989

Cited by 7 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Decreased serum levels of sCD40L and IL-31 correlate in treated patients with Relapsing-Remitting Multiple Sclerosis;Immunobiology;2018-01

2. Minocycline plus N-acteylcysteine induces remyelination, synergistically protects oligodendrocytes and modifies neuroinflammation in a rat model of mild traumatic brain injury;Journal of Cerebral Blood Flow & Metabolism;2017-07-07

3. Inhibition of CD40-TRAF6 interactions by the small molecule inhibitor 6877002 reduces neuroinflammation;Journal of Neuroinflammation;2017-05-12

4. Whole-exome sequence analysis of ataxia telangiectasia-like phenotype;Journal of the Neurological Sciences;2014-05

5. Establishment of a Model to Examine the Early Events Involved in the Development of Virus-Induced Demyelinating Lesions;Annals of the New York Academy of Sciences;2007-03-21

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