Association of EGFR Gene Amplification and CDKN2 (p16/MTS1) Gene Deletion in Glioblastoma Multiforme-Reference-Cited by-同舟云学术

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Association of EGFR Gene Amplification and CDKN2 (p16/MTS1) Gene Deletion in Glioblastoma Multiforme

Published:1997-07 Issue:3 Volume:7 Page:871-875
ISSN:1015-6305
Container-title:Brain Pathology
language:en
Short-container-title:Brain Pathology

Author:

Hayashi Yutaka,Ueki Keisuke,Waha Andreas,Wiestler Otmar D.,Louis David N.,Deimlina Andreas

Publisher

Wiley

Subject

Neurology (clinical),Pathology and Forensic Medicine,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1750-3639.1997.tb00890.x/fullpdf

Reference29 articles.

1. Tumorigenicity and Gene Amplification Potentials of Cyclin D1-Overexpressing NIH3T3 Cells

2. Gene Amplification in Malignant Human Gliomas: Clinical and Histopathologic Aspects

3. Patient age, histologic features, and length of survival in patients with glioblastoma multiforme

4. Double minute chromosomes can be produced from precursors derived from a chromosomal deletion.

5. Wild-type p53 mediates apoptosis by E1A, which is inhibited by E1B.

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2. EGFR suppresses p53 function by promoting p53 binding to DNA-PKcs: a noncanonical regulatory axis between EGFR and wild-type p53 in glioblastoma;Neuro-Oncology;2022-04-26

3. Current understanding of gliomagenesis: from model to mechanism;International Journal of Medical Sciences;2022

4. Clonal Evolution of a High-Grade Pediatric Glioma With Distant Metastatic Spread;Neurology Genetics;2021-02-15

5. Characterization of molecular pathways for targeting therapy in glioblastoma;Chinese Clinical Oncology;2020-12

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