Increased ferritin levels in non‐transfusion‐dependent β°‐thalassaemia/HbE are associated with reduced CXCR2 expression and neutrophil migration-Reference-Cited by-同舟云学术

Increased ferritin levels in non‐transfusion‐dependent β°‐thalassaemia/HbE are associated with reduced CXCR2 expression and neutrophil migration

Published:2019-12-29 Issue:1 Volume:189 Page:187-198
ISSN:0007-1048
Container-title:British Journal of Haematology
language:en
Short-container-title:Br J Haematol

Author:

Thiengtavor Chayada¹²,Siriworadetkun Sirikwan¹³,Paiboonsukwong Kittiphong³,Fucharoen Suthat³,Pattanapanyasat Kovit⁴,Vadolas Jim⁵⁶,Svasti Saovaros³⁷^ORCID,Chaichompoo Pornthip¹^ORCID

Affiliation:

1. Department of Pathobiology Faculty of Science Mahidol University Bangkok Thailand

2. Faculty of Optometry Ramkhamhaeng University Bangkok Thailand

3. Thalassemia Research Center Institute of Molecular Biosciences Mahidol University Nakhon Pathom Thailand

4. Siriraj Center of Research Excellence for Microparticle and Exosome in Diseases Faculty of Medicine Siriraj Hospital Mahidol University Bangkok Thailand

5. Centre for Cancer Research Hudson Institute of Medical Research Melbourne Australia

6. Department of Molecular and Translational Science Monash University Melbourne Australia

7. Department of Biochemistry Faculty of Science Mahidol University Bangkok Thailand

Funder

Thailand Research Fund

Office of the Higher Education Commission

Mahidol University

National Research Council of Thailand

Publisher

Wiley

Subject

Hematology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjh.16295

Reference43 articles.

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2. Intravascular Hemolysis, Vascular Endothelial Cell Activation and Thrombophilia in Splenectomized Patients with Hemoglobin E/β-Thalassemia Disease

3. Neutrophils migrate across intestinal epithelium using β2 integrin (CD11b/CD18)-independent mechanisms

4. Dose effects of LPS on neutrophils- in a whole blood flow cytometric assay of phagocytosis and oxidative burst

5. Complement Receptor 3 Deficiency Influences Lesion Progression during Leishmania major Infection in BALB/c Mice

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