JAK2‐STAT3 signaling pathway is involved in rat periapical lesions induced by Enterococcus faecalis
Author:
Affiliation:
1. Department of Endodontics and Periodontics, College of Stomatology Dalian Medical University Dalian China
2. Department of Oral Basic science, College of Stomatology Dalian Medical University Dalian China
Publisher
Wiley
Subject
General Dentistry,Otorhinolaryngology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/odi.13169
Reference38 articles.
1. Lipoteichoic Acid Partially Contributes to the Inflammatory Responses to Enterococcus faecalis
2. Activation of the Acquired Immune Response Reduces Coupled Bone Formation in Response to a Periodontal Pathogen
3. Requirement for JAK2 inErythropoietin-Induced Signalling Pathways
4. The JAK inhibitor tofacitinib suppresses synovial JAK1-STAT signalling in rheumatoid arthritis
5. STAT3 activity is necessary and sufficient for the development of immune‐mediated myocarditis in mice and promotes progression to dilated cardiomyopathy
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1. Rev‐erbα attenuates refractory periapical periodontitis via M1 polarization: An in vitro and in vivo study;International Endodontic Journal;2024-01-27
2. Phage PEf771 for the Treatment of Periapical Periodontitis Induced by Enterococcus faecalis YN771;Critical Reviews in Immunology;2024
3. A novel experimental model to investigate fungal involvement shows expression of Dectin‐1 in periapical lesion pathogenesis;Journal of Oral Rehabilitation;2023-06-09
4. MicroRNA-146b-5p Suppresses Pro-Inflammatory Mediator Synthesis via Targeting TRAF6, IRAK1, and RELA in Lipopolysaccharide-Stimulated Human Dental Pulp Cells;International Journal of Molecular Sciences;2023-04-18
5. Role of Enterococcus faecalis in refractory apical periodontitis: from pathogenicity to host cell response;Journal of Oral Microbiology;2023-03
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