Two novel antitheticalKNblood group antigens may contribute to more than a quarter of allKNantisera inEurope-Reference-Cited by-同舟云学术

Two novel antitheticalKNblood group antigens may contribute to more than a quarter of allKNantisera inEurope

Published:2020-09 Issue:10 Volume:60 Page:2408-2418
ISSN:0041-1132
Container-title:Transfusion
language:en
Short-container-title:Transfusion

Author:

Grueger Daniela¹²,Zeretzke Anja¹,Habicht Cora P.¹,Skaik Younis¹,Wagner Franz F.³⁴^ORCID,Scharberg Erwin A.⁵,Costelloe Aisling⁶,Martens Jörg²,Verboom Murielle²,Bugert Peter⁷^ORCID,Schneeweiss Clemens¹^ORCID

Affiliation:

1. Imusyn GmbH & Co. KG Hanover Germany

2. Institute of Transfusion Medicine and Transplant Engineering Hannover Medical School Hanover Germany

3. German Red Cross Blood Service NSTOB Springe Germany

4. MVZ am Clementinenkrankenhaus Springe Germany

5. Institute of Transfusion Medicine and Immunohematology German Red Cross Blood Service Baden‐Württemberg ‐ Hessen Baden‐Baden Germany

6. RCI Laboratory, National Blood Center Irish Blood Transfusion Service Dublin Ireland

7. Institute of Transfusion Medicine and Immunology, Medical Faculty Mannheim Heidelberg University, German Red Cross Blood Service Baden‐Württemberg – Hessen gGmbH Mannheim Germany

Publisher

Wiley

Subject

Hematology,Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/trf.16039

Reference18 articles.

1. International Society of Blood Transfusion Working Party on Red Cell Immunogenetics and Blood Group Terminology: Report of the Dubai, Copenhagen and Toronto meetings

2. The C3b/C4b receptor is recognized by the Knops, McCoy, Swain-langley, and York blood group antisera.

3. Identification of distinct C3b and C4b recognition sites in the human C3b/C4b receptor (CR1, CD35) by deletion mutagenesis.

4. Analysis of the functional domains of complement receptor type 1 (C3b/C4b receptor; CD35) by substitution mutagenesis;Krych M;J Biol Chem,1994

5. The clearance of tetanus toxoid/anti‐tetanus toxoid immune complexes from the circulation of humans. Complement‐ and erythrocyte complement receptor 1‐dependent mechanisms;Schifferli JA;J Immunol,1988

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