NOD1 is required forHelicobacter pyloriinduction of IL-33 responses in gastric epithelial cells-Reference-Cited by-同舟云学术

NOD1 is required forHelicobacter pyloriinduction of IL-33 responses in gastric epithelial cells

Published:2018-02-15 Issue:5 Volume:20 Page:e12826
ISSN:1462-5814
Container-title:Cellular Microbiology
language:en
Short-container-title:Cellular Microbiology

Author:

Tran Le Son¹,Tran Darren¹,De Paoli Amanda¹,D'Costa Kimberley¹,Creed Sarah J.²,Ng Garrett Z.³⁴,Le Lena¹,Sutton Philip³⁴⁵,Silke J.⁶⁷,Nachbur U.⁶⁷,Ferrero Richard L.¹⁸^ORCID

Affiliation:

1. Centre for Innate Immunity and Infectious Diseases, The Hudson Institute of Medical Research; Monash University; Clayton Victoria Australia

2. Monash Micro Imaging, The Hudson Institute of Medical Research; Monash University; Clayton Victoria Australia

3. Murdoch Children's Research Institute; The Royal Children's Hospital; Parkville Victoria Australia

4. School of Veterinary and Agricultural Science; The University of Melbourne; Parkville Victoria Australia

5. Department of Paediatrics; The University of Melbourne; Parkville Victoria Australia

6. Division of Cell Signalling and Cell Death; The Walter and Eliza Hall Institute; Parkville Victoria Australia

7. Department of Medical Biology; The University of Melbourne; Parkville Victoria Australia

8. Biomedicine Discovery Institute, Department of Microbiology; Monash University; Clayton Victoria Australia

Funder

National Health and Medical Research Council

Faculty of Medicine, Nursing and Health Sciences, Monash University

Publisher

Hindawi Limited

Subject

Virology,Immunology,Microbiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/cmi.12826/fullpdf

Reference46 articles.

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2. Posttranslational modification as a critical determinant of cytoplasmic innate immune recognition;Baker;Physiological Reviews,2017

3. Rapid development of severe hyperplastic gastritis with gastric epithelial dedifferentiation in Helicobacter felis-infected IL-10(−/−) mice;Berg;The American Journal of Pathology,1998

4. Helicobacter pylori cag pathogenicity island (cagPAI) involved in bacterial internalization and IL-8 induced responses via NOD1- and MyD88-dependent mechanisms in human biliary epithelial cells;Boonyanugomol;PLoS One,2013

5. IL33 is a stomach alarmin that initiates a skewed Th2 response to injury and infection;Buzzelli;Cellular and Molecular Gastroenterology and Hepatology,2015

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