High fat programming of beta cell compensation, exhaustion, death and dysfunction-Reference-Cited by-同舟云学术

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High fat programming of beta cell compensation, exhaustion, death and dysfunction

Published:2014-04-03 Issue:2 Volume:16 Page:71-78
ISSN:1399-543X
Container-title:Pediatric Diabetes
language:en
Short-container-title:Pediatr Diabetes

Author:

Cerf Marlon E.¹

Affiliation:

1. Diabetes Discovery Platform; Medical Research Council; Tygerberg Cape Town South Africa

Publisher

Wiley

Subject

Endocrinology, Diabetes and Metabolism,Pediatrics, Perinatology, and Child Health,Internal Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/pedi.12137/fullpdf

Reference62 articles.

1. Beta cell differentiation during early human pancreas development;Piper;J Endocrinol,2004

2. TGF-beta plays a key role in morphogenesis of the pancreatic islets of Langerhans by controlling the activity of the matrix metalloproteinase MMP-2;Miralles;J Cell Biol,1998

3. Growth dynamics of pancreatic islet cell populations during fetal and neonatal development of the rat;Kaung;Dev Dyn,1994

4. Inflammation is necessary for long-term but not short-term high-fat diet-induced insulin resistance;Lee;Diabetes,2011

5. Chronic inflammation exacerbates glucose metabolism disorders in C57BL/6J mice fed with high-fat diet;Wu;J Endocrinol,2013

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5. Effectiveness of Hydroalcoholic Seed Extract of Securigera securidaca on Pancreatic Local Renin-Angiotensin System and Its Alternative Pathway in Streptozotocin-Induced Diabetic Animal Model;Oxidative Medicine and Cellular Longevity;2023-01-07

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