C9 ORF 72 expansion in a family with bipolar disorder-Reference-Cited by-同舟云学术

C9 ORF 72 expansion in a family with bipolar disorder

Published:2013-04 Issue:3 Volume:15 Page:326-332
ISSN:1398-5647
Container-title:Bipolar Disorders
language:en
Short-container-title:Bipolar Disord

Author:

Meisler Miriam H¹²,Grant Adrienne E¹,Jones Julie M¹,Lenk Guy M¹,He Fang²,Todd Peter K²,Kamali Masoud³⁴,Albin Roger L²⁵⁶,Lieberman Andrew P⁶⁷,Langenecker Scott A³⁴,McInnis Melvin G³⁴

Affiliation:

1. Department of Human Genetics University of Michigan School of MedicineAnn Arbor MI USA

2. Department of Neurology University of Michigan School of MedicineAnn Arbor MI USA

3. Department of Psychiatry University of Michigan School of MedicineAnn Arbor MI USA

4. University of Michigan Depression CenterAnn Arbor MI USA

5. Geriatrics Research Education and Clinical Center VAAAHSAnn Arbor MI USA

6. Michigan Alzheimer Disease CenterAnn Arbor MI USA

7. Department of Pathology University of Michigan Ann Arbor MI USA

Publisher

Wiley

Subject

Biological Psychiatry,Psychiatry and Mental health

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bdi.12063

Reference22 articles.

1. A Hexanucleotide Repeat Expansion in C9ORF72 Is the Cause of Chromosome 9p21-Linked ALS-FTD

2. A C9orf72 promoter repeat expansion in a Flanders-Belgian cohort with disorders of the frontotemporal lobar degeneration-amyotrophic lateral sclerosis spectrum: a gene identification study

3. Expanded GGGGCC Hexanucleotide Repeat in Noncoding Region of C9ORF72 Causes Chromosome 9p-Linked FTD and ALS

4. The C9ORF72 expansion mutation is a common cause of ALS+/−FTD in Europe and has a single founder

5. Frequency of the C9orf72 hexanucleotide repeat expansion in patients with amyotrophic lateral sclerosis and frontotemporal dementia: a cross-sectional study

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