Increased Arf/p53 activity in stem cells, aging and cancer-Reference-Cited by-同舟云学术

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Increased Arf/p53 activity in stem cells, aging and cancer

Published:2017-01-19 Issue:2 Volume:16 Page:219-225
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Carrasco-Garcia Estefania¹,Moreno Manuel¹,Moreno-Cugnon Leire¹,Matheu Ander¹²

Affiliation:

1. Cellular Oncology Group; Biodonostia Institute; San Sebastian Spain

2. Ikerbasque; Basque Foundation; Bilbao Spain

Funder

Eusko Jaurlaritza

Instituto de Salud Carlos III

Federación Española de Enfermedades Raras

Diputacion Guipuzcoa

Marie Curie Career Integration

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acel.12574/fullpdf

Reference68 articles.

1. The ataxia telangiectasia-mutated target site Ser18 is required for p53-mediated tumor suppression;Armata;Cancer Res.,2007

2. Opposing roles for p16Ink4a and p19Arf in senescence and ageing caused by BubR1 insufficiency;Baker;Nat. Cell Biol.,2008

3. Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders;Baker;Nature,2011

4. p21 both attenuates and drives senescence and aging in BubR1 progeroid mice;Baker;Cell Rep.,2013

5. Naturally occurring p16(Ink4a)-positive cells shorten healthy lifespan;Baker;Nature,2016

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