Clinical correlates of submicroscopic deletions involving the ABL-BCR translocation region in chronic myeloid leukemia
Author:
Publisher
Wiley
Subject
Hematology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0609.2004.00356.x/fullpdf
Reference13 articles.
1. Chronic Myelogenous Leukemia
2. The diversity of BCR-ABL fusion proteins and their relationship to leukemia phenotype [editorial; comment]
3. Double jeopardy from a single translocation: deletions of the derivative chromosome 9 in chronic myeloid leukemia
4. Large deletions at the t(9;22) breakpoint are common and may identify a poor-prognosis subgroup of patients with chronic myeloid leukemia
5. Deletions of the derivative chromosome 9 occur at the time of the Philadelphia translocation and provide a powerful and independent prognostic indicator in chronic myeloid leukemia
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1. Entire ABL1 Gene Deletion Without BCR/ABL1 Rearrangement in a Female Patient with B-Cell Precursor Acute Lymphoblastic Leukemia;OncoTargets and Therapy;2020-01
Entire ABL1 Gene Deletion Without BCR/ABL1 Rearrangement in a Female Patient with B-Cell Precursor Acute Lymphoblastic Leukemia
2. Prognostic Implications of Derivative Chromosome 9 Deletions in Patients with Advanced-Stage Chronic Myelogenous Leukemia;Journal of Environmental Pathology, Toxicology and Oncology;2018
3. Chronic myeloid leukemia;Cancer Cytogenetics;2015-06-26
4. Chronic Myelogenous Leukemia;Current Cancer Therapy Reviews;2012-02-01
5. Genome-wide high density single-nucleotide polymorphism array-based karyotyping improves detection of clonal aberrations including der(9) deletion, but does not predict treatment outcomes after imatinib therapy in chronic myeloid leukemia;Annals of Hematology;2011-03-08
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