Postelicitation model of allergic contact dermatitis for predicting the efficacy of topical drugs
Author:
Publisher
Wiley
Subject
Dermatology,Molecular Biology,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0625.2008.00757.x/fullpdf
Reference22 articles.
1. Analyses of a mouse model of the dermatitis caused by TNCB-repeated application;Harada;J Dermatol Sci,2005
2. Oral administration of Astragalus membranaceus inhibits the development of DNFB-induced dermatitis in NC/Nga mice;Lee;Biol Pharm Bull,2007
3. Pharmacological profile of a novel phosphodiesterase 7A and -4 dual inhibitor, YM-393059, on acute and chronic inflammation models;Yamamoto;Eur J Pharmacol,2006
4. Non-steroidal and steroidal anti-inflammatory drugs vary in their modulation of dendritic cell function in the elicitation phase of allergic contact dermatitis;Bäumer;Exp Dermatol,2006
5. Effect of topical cis-urocanic acid on lymph node activation during contact sensitization in mouse, rat and guinea pig;Lauerma;Br J Dermatol,1996
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1. Observation of hapten-induced sensitization responses for the development of a mouse skin sensitization test, including the elicitation phase;Scientific Reports;2022-11-18
2. Contact dermatitis: Allergic and irritant;Clinics in Dermatology;2014-01
3. Anti-Inflammatory Effects of Urocanic Acid Derivatives in Models Ex Vivo and In Vivo of Inflammatory Bowel Disease;ISRN Inflammation;2012-09-05
4. Extended DNFB-induced contact hypersensitivity models display characteristics of chronic inflammatory dermatoses;Experimental Dermatology;2011-12-12
5. New and emerging cosmetic allergens;Clinics in Dermatology;2011-05
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