The Biochemical and Clinical Consequences of 2′-Deoxycoformycin in T Cell Chronic Lymphocytic Leukaemia
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0609.1984.tb00678.x/fullpdf
Reference30 articles.
1. Adenosine deaminase deficiency in two patients with severely impaired cellular immunity;Giblett;Lancet,1972
2. Inhibition of adenosine deaminase activity results in cytotoxicity to T lymphoblasts in vivo;Mitchell;Blood,1980
3. Therapeutic selectivity and prediction of response to 2′-deoxycoformycin in acute leukaemia;Prentice;Lancet,1981
4. Deoxyadenosine triphosphate as a potentially toxic metabolite in adenosine deaminase deficiency;Cohen;Proc Natl Acad Sci USA,1978
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Induction of apoptosis in CD4+ prolymphocytic leukemia by deoxyadenosine and 2′-deoxycoformycin;Leukemia Research;1992-08
2. Deoxycoformycin treatment for childhood T-cell acute lymphoblastic leukemia early in second remission: A pediatric oncology group study;Medical and Pediatric Oncology;1988
3. Biochemical consequences of 2′-deoxycoformycin treatment in a patient with T-cell lymphoma. Some unusual findings;Cancer;1987-08-15
4. Differential metabolism of deoxyribonucleosides by leukaemic T cells of immature and mature phenotype;British Journal of Haematology;1985-09
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