Experimental arthritis exacerbates Aggregatibacter actinomycetemcomitans-induced periodontitis in mice

Author:

Queiroz-Junior Celso Martins,Madeira Mila Fernandes Moreira,Coelho Fernanda Matos1,Oliveira Camila Ribeiro,Cândido Luíza Castro Menezes1,Garlet Gustavo Pompermaier2,Teixeira Mauro Martins1,Souza Daniele da Glória3,Silva Tarcília Aparecida da

Affiliation:

1. Department of Biochemistry and Immunology, Instituto de Ciências Biológicas; Universidade Federal de Minas Gerais; Minas Gerais; Brazil

2. Department of Biological Sciences; School of Dentistry of Bauru, Universidade de São Paulo; São Paulo; Brazil

3. Department of Microbiology, Instituto de Ciências Biológicas; Universidade Federal de Minas Gerais; Minas Gerais; Brazil

Publisher

Wiley

Subject

Periodontics

Reference37 articles.

1. Dipeptidyl peptidase I activates neutrophil-derived serine proteases and regulates the development of acute experimental arthritis;Adkison;Journal of Clinical Investigation,2002

2. Epidemiology and risk factors of periodontal diseases;Albandar;Dental Clinics of North America,2005

3. IL-1 and TNF antagonists inhibit the inflammatory response and bone loss in experimental periodontitis;Assuma;The Journal of Immunology,1998

4. Impaired inflammatory angiogenesis, but not leukocyte influx, in mice lacking TNFR1;Barcelos;Journal of Leukocyte Biology,2005

5. Effect of Porphyromonas gingivalis-induced inflammation on the development of rheumatoid arthritis;Bartold;Journal of Clinical Periodontology,2010

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