Effects of interleukin 1β, tumor necrosis factor α and transforming growth factor β on group II phospholipase A2activity in rat gingival fibroblasts
Author:
Publisher
Wiley
Subject
Periodontics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0765.1993.tb02116.x/fullpdf
Reference17 articles.
1. Intracellular phospholipase A;Boch;Biochim Biophys Acta,1980
2. Serum phospholipase A2 correlates with disease activity in rheumatoid arthritis;Pruzanskji;J Rheumatol,1988
3. The role of phospholipase A in acute pancreatitis;Nevalainen;Scand J Gastroenterol,1980
4. Extracellular phospholipase A2: causative agent in circulatory collapse of septic shock;Vadas;Agents Actions,1988
5. Raised serum activity of phospholipase A2 immunochemically related to group II enzyme in inflammatory bowel disease: its correlation with disease activity of Crohn's disease and ulcerative colitis;Minami;Gut,1992
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1. Correlation of Levels of Interleukin-1β in Gingival Crevicular Fluid to the Clinical Parameters of Chronic Periodontitis;The Journal of Contemporary Dental Practice;2011
2. Measurement of prostaglandin E2 and leukotriene B4 in the gingival crevicular fluid;Journal of Dentistry;1998-03
3. Mepacrine decreases lung leak in rats given interleukin-1 intratracheally.;American Journal of Respiratory and Critical Care Medicine;1997-05
4. Cyclosporin A upregulates prostaglandin E2 production in human gingival fibroblasts challenged with tumor necrosis factor alpha in vitro;Journal of Oral Pathology and Medicine;1997-01
5. Periodontal Diseases: Pathogenesis;Annals of Periodontology;1996-11
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