Experimental periodontitis in mice selected for maximal or minimal inflammatory reactions: increased inflammatory immune responsiveness drives increased alveolar bone loss without enhancing the control of periodontal infection

Author:

Trombone A. P. F.,Ferreira Jr S. B.,Raimundo F. M.,de Moura K. C. R.,Avila-Campos M. J.,Silva J. S.,Campanelli A. P.,De Franco M.,Garlet G. P.

Publisher

Wiley

Subject

Periodontics

Reference35 articles.

1. The dual role of p55 tumour necrosis factor-alpha receptor in Actinobacillus actinomycetemcomitans-induced experimental periodontitis: host protection and tissue destruction;Garlet;Clin Exp Immunol,2007

2. Immune response: the key to bone resorption in periodontal disease;Taubman;J Periodontol,2005

3. Clinical benefits of oral irrigation for periodontitis are related to reduction of pro-inflammatory cytokine levels and plaque;Cutler;J Clin Periodontol,2000

4. The contribution of interleukin-1 and tumor necrosis factor to periodontal tissue destruction;Graves;J Periodontol,2003

5. Balance of inflammatory response in stable gingivitis and progressive periodontitis lesions;Honda;Clin Exp Immunol,2006

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