Cigarette smoke condensate stimulates urokinase production through the generation of reactive oxygen species and activation of the mitogen activated protein kinase pathways in human gingival fibroblasts
Author:
Publisher
Wiley
Subject
Periodontics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0765.2008.01114.x/fullpdf
Reference50 articles.
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2. Tobacco smoking and risk for periodontal disease;Bergstrom;J Clin Periodontol,2003
3. The impact of cigarette smoking on periodontal disease and treatment;Johnson;Periodontol 2000,2007
4. Smoking interferes with the prognosis of dental implant treatment: a systematic review and meta-analysis;Strietzel;J Clin Periodontol,2007
5. Molecular and cell biology of the gingiva;Bartold;Periodontol 2000,2000
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2. Role of mitogen-activated protein kinase pathways in migration of gingival epithelial cells in response to stimulation by cigarette smoke condensate and infection by Porphyromonas gingivalis;Journal of Periodontal Research;2015-12-15
3. Interaction of gingival epithelial cells with periodontal pathogen under cigarette smoke stimulation;Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology);2015
4. Cigarette smoke condensate modulates migration of human gingival epithelial cells and their interactions with Porphyromonas gingivalis;Journal of Periodontal Research;2014-09-05
5. Cigarette sidestream smoke induces histone H3 phosphorylation via JNK and PI3K/Akt pathways, leading to the expression of proto-oncogenes;Carcinogenesis;2014-01-07
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