Mutational analysis of PIK3CA,JAK2,BRAF,FOXL2,IDH1,AKT1 and EZH2 oncogenes in sarcomas

Author:

Je Eun M.1,An Chang H.2,Yoo Nam J.1,Lee Sug H.

Affiliation:

1. Department of Pathology; College of Medicine; The Catholic University of Korea; Seoul; Korea

2. Department of General Surgery; College of Medicine; The Catholic University of Korea; Seoul; Korea

Publisher

Wiley

Subject

Microbiology (medical),General Medicine,Immunology and Allergy,Pathology and Forensic Medicine

Reference20 articles.

1. Soft tissue sarcomas of adults: state of the translational science;Borden;Clin Cancer Res,2003

2. Subtype-specific genomic alterations define new targets for soft-tissue sarcoma therapy;Barretina;Nat Genet,2010

3. Differential transactivation by alternative EWS-FLI1 fusion proteins correlates with clinical heterogeneity in Ewing's sarcoma;Lin;Cancer Res,1999

4. The PAX3-FKHR fusion protein created by the t(2;13) translocation in alveolar rhabdomyosarcomas is a more potent transcriptional activator than PAX3;Fredericks;Mol Cell Biol,1995

5. Fusion of the dominant negative transcription regulator CHOP with a novel gene FUS by translocation t(12;16) in malignant liposarcoma;Rabbitts;Nat Genet,1993

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