Calsyntenins Mediate TGN Exit of APP in a Kinesin-1-Dependent Manner-Reference-Cited by-同舟云学术

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Calsyntenins Mediate TGN Exit of APP in a Kinesin-1-Dependent Manner

Published:2009-05 Issue:5 Volume:10 Page:572-589
ISSN:1398-9219
Container-title:Traffic
language:en
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Author:

Ludwig Alexander,Blume Jessica,Diep Tu-My,Yuan Ju,Mateos José María,Leuthäuser Kerstin,Steuble Martin,Streit Peter,Sonderegger Peter

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0854.2009.00886.x/fullpdf

Reference53 articles.

1. The trans-Golgi network: a late secretory sorting station;Traub;Curr Opin Cell Biol,1997

2. Domains of the TGN: coats, tethers and G proteins;Gleeson;Traffic,2004

3. Exiting the Golgi complex;De Matteis;Nat Rev Cancer,2008

4. Kinesin and dynein superfamily proteins and the mechanism of organelle transport;Hirokawa;Science,1998

5. Motoring around the Golgi;Allan;Nat Cell Biol,2002

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1. Calsyntenin‐1 expression and function in brain tissue of lithium–pilocarpine rat seizure models;Synapse;2024-08-22

2. Suppression of the amyloidogenic metabolism of APP and the accumulation of Aβ by alcadein α in the brain during aging;Scientific Reports;2024-08-09

3. Unveiling New Genetic Variants Associated with Age at Onset in Alzheimer’s Disease and Frontotemporal Lobar Degeneration Due to C9orf72 Repeat Expansions;International Journal of Molecular Sciences;2024-07-07

4. MAP4 kinase-regulated reduced CLSTN1 expression in medulloblastoma is associated with increased invasiveness;2024-03-21

5. Axonal transport of Frizzled5 by Alcadein α-containing vesicles is associated with kinesin-1;Molecular Biology of the Cell;2023-10-01

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