The Use of Flufenamate and Forskolin to Evaluate the Role of cAMP in Gonadotropin-Releasing Hormone-Stimulated Luteinizing Hormone Secretion from Pituitaries of Ovariectomized Rats-Reference-Cited by-同舟云学术

The Use of Flufenamate and Forskolin to Evaluate the Role of cAMP in Gonadotropin-Releasing Hormone-Stimulated Luteinizing Hormone Secretion from Pituitaries of Ovariectomized Rats

Published:1992-11 Issue:5 Volume:71 Page:395-400
ISSN:0901-9928
Container-title:Pharmacology & Toxicology
language:en
Short-container-title:

Author:

Das Saswati,Bourne Gregory A.

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Pharmacology,Toxicology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0773.1992.tb00569.x/fullpdf

Reference26 articles.

1. Characterization of LH responses to continuous infusions of GnRH using perifused pituitaries from intact, ovariectominzed and steroid-treated rats;Baldwin;Biol. Reprod.,1983

2. Cyclic AMP indirectly mediates the extracellular Ca++-independent release of LH;Bourne;Mol. Cell. Endocrinol.,1988

3. Forskolin but not cholera toxin bypasses flufenamate-induced inhibition of cAMP production in anterior pituitaries;Bourne;Pharmacology & Toxicology,1992

4. Extracellular Ca++-independent and -dependent components of the biphasic release of LH in response to luteinizing hormone-releasing hormone in vitro;Bourne;Endocrinology,1980

5. Evidence for cAMP as a mediator of gonadotropin secretion from female pituitaries;Bourne;Amer. J. Physiol.,1987

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1. Forskolin and derivatives as tools for studying the role of cAMP;PHARMAZIE;2012

2. Effect of cAMP on GnRH stimulated LH secretion from individual pituitary gonadotropes;Molecular and Cellular Endocrinology;1995-10-30

3. Mechanism of melatonin signal transduction in the neonatal rat pituitary;Neurochemistry International;1995-09

4. Melatonin inhibition of GnRH-induced LH release from neonatal rat gonadotroph: involvement of Ca2+ not cAMP;American Journal of Physiology-Endocrinology and Metabolism;1995-07-01

5. Adenosine 3′,5′-cyclic monophosphate and the self-priming effect of gonadotrophin-releasing hormone;Molecular and Cellular Endocrinology;1995-01