Glutamate and aspartate are decreased in the skin in amyotrophic lateral sclerosis
Author:
Publisher
Wiley
Subject
Neurology (clinical),Neurology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0404.1992.tb05128.x/fullpdf
Reference19 articles.
1. A remarkable absence of decubitus formation in patients with amyotrophic lateral sclerosis;Toyokura;Neurol Med Chir (Tokyo),1975
2. Amyotrophic lateral sclerosis: a clinical and pathological study on the “negative features” of the disease;Toyokura;Jpn J Med,1977
3. Prognosis of motor neuron disease in Japan;Uebayashi;Neuroepidemiology,1983
4. Amyotrophic lateral sclerosis: histologic, histochemical, and ultra-structural abnormalities of skin;Ono;Neurology,1986
5. “Delayed return phenomenon” in amyotrophic lateral sclerosis;Ono;Acta Neurol Scand,1988
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1. An explanation for the changes in collagen in sporadic Amyotrophic Lateral Sclerosis;Medical Hypotheses;2011-10
2. MMP-2 and MMP-9 are elevated in spinal cord and skin in a mouse model of ALS;Journal of the Neurological Sciences;2010-07
3. Reply;Acta Neurologica Scandinavica;2009-01-29
4. Excitatory amino acids and amyotrophic lateral sclerosis;Acta Neurologica Scandinavica;2009-01-29
5. Leukemia Inhibitory Factor Induces Epidermal Hyperplasia in Patients with Amyotrophic Lateral Sclerosis;Journal of Investigative Dermatology;2000-09
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