Effects of Acute Ketotic and Non-ketotic Diabetes on the Myocardial Muscarinic Receptors
Author:
Publisher
Wiley
Subject
Pharmacology,Toxicology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0773.1984.tb01997.x/fullpdf
Reference17 articles.
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2. Relationship between the inhibition constant (K1) and the concentration of inhibitor which causes 50 per cent inhibition (I50) of an enzymatic reaction;Cheng;Biochem. Pharmacol.,1973
3. Guanine nucleotide binding regulatory proteins and dual control of adenylate cyclase;Gilman;J. Clin. Invest.,1984
4. Proteolytic activation of rat liver adenylate cyclase by a contaminant of collagenase from Clostridium histolyticum;Hanoune;J. Biol. Chem.,1977
5. Differential modification of the interaction of cardiac muscarinic cholinergic and beta-adrenergic receptors with a guanine nucleotide binding component;Harden;Mol. Pharmacol.,1982
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1. Adenylate Cyclase Modulation by Ammonium Ion: GTP-like Effect on Muscarinic and α2-Adrenergic Receptors;Acta Pharmacologica et Toxicologica;2009-03-13
2. Increased Expression of Gi-Coupled Muscarinic Acetylcholine Receptor and Gi in Atrium of Elderly Diabetic Subjects;Diabetes;2004-09-01
3. Increased function of inhibitory neuronal M2muscarinic receptors in diabetic rat lungs;British Journal of Pharmacology;1997-07
4. Diabetes-Induced Changes in the Gi-Modulated Muscarinic Receptor-Adenylyl Cyclase System in Rat Myocardium;Pharmacology & Toxicology;1994-09
5. Parotid Gland Function in Streptozotocin-diabetic Rats;Journal of Dental Research;1987-02
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