Renal tubular expression of Toll-like receptor 4 in cyclosporine nephrotoxicity
Author:
Publisher
Wiley
Subject
Microbiology (medical),General Medicine,Immunology and Allergy,Pathology and Forensic Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0463.2009.02514.x/fullpdf
Reference43 articles.
1. Follow-up study of children with nephrotic syndrome treated with a long-term moderate dose of cyclosporine;Hino;Am J Kidney Dis,1998
2. Cyclosporine nephrotoxicity;Burdmann;Semin Nephrol,2003
3. Cyclosporine nephrotoxicity: how does it affect renal allograft function and transplant morphology?;Morozumi;Transplant Proc,2004
4. Primer: histopathology of calcineurin-inhibitor toxicity in renal allografts;Liptak;Nat Clin Pract Nephrol,2006
5. Interaction of cyclosporine A and the renin-angiotensin system; new perspectives;Lassila;Curr Drug Metab,2002
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1. Oxygenation of the Transplanted Kidney;Seminars in Nephrology;2019-11
2. TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity;Archives of Toxicology;2016-09-01
3. Renal expression of Toll-like receptor 2 and 4: Dynamics in human allograft injury and comparison to rodents;Molecular Immunology;2015-03
4. Toll-like receptors: sensing and reacting to diabetic injury in the kidney;Nephrology Dialysis Transplantation;2013-11-07
5. Calcineurin inhibitors recruit protein kinases JAK2 and JNK, TLR signaling and the UPR to activate NF-κB-mediated inflammatory responses in kidney tubular cells;Toxicology and Applied Pharmacology;2013-11
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