Chronic L‐ DOPA induces hyperactivity, normalization of gait and dyskinetic behavior in MitoPark mice-Reference-Cited by-同舟云学术

Chronic L‐ DOPA induces hyperactivity, normalization of gait and dyskinetic behavior in MitoPark mice

Published:2015-03 Issue:3 Volume:14 Page:260-270
ISSN:1601-1848
Container-title:Genes, Brain and Behavior
language:en
Short-container-title:Genes, Brain and Behavior

Author:

Gellhaar S.¹,Marcellino D.¹²,Abrams M. B.¹,Galter D.¹

Affiliation:

1. Department of Neuroscience Karolinska Institute Stockholm Sweden

2. Present address: Department of Physiology and Institute of Biomedical Technology (ITB), Center for Biomedical Research of the Canary Islands (CIBICAN) University of La Laguna School of Medicine Tenerife Spain

Funder

Swedish Brain Power

Swedish Research Council

Publisher

Wiley

Subject

Behavioral Neuroscience,Neurology,Genetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/gbb.12210

Reference27 articles.

1. Gait dynamics in mouse models of Parkinson's disease and Huntington's disease

2. Striatal fosB Expression Is Causally Linked with l-DOPA-Induced Abnormal Involuntary Movements and the Associated Upregulation of Striatal Prodynorphin mRNA in a Rat Model of Parkinson's Disease

3. Dopa-sensitive and Dopa-resistant gait parameters in Parkinson's disease

4. Blumstein D.T. Daniel J.C. Evans C.S.(2006)JWatcher 1.0: An Introductory User's Guide (Computer software). University of California Los Angeles CA.

5. L-DOPA-induced dyskinesia in the rat is associated with striatal overexpression of prodynorphin- and glutamic acid decarboxylase mRNA

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