IL ‐22–expressing CD 4 + AhR + T lymphocytes are associated with RANKL ‐mediated alveolar bone resorption during experimental periodontitis

Author:

Monasterio Gustavo1ORCID,Budini Victoria1ORCID,Fernández Baltasar1ORCID,Castillo Francisca1ORCID,Rojas Carolina1ORCID,Alvarez Carla12ORCID,Cafferata Emilio A.13ORCID,Vicencio Emiliano45ORCID,Cortés Bastián I.45ORCID,Cortez Cristian5ORCID,Vernal Rolando16ORCID

Affiliation:

1. Faculty of DentistryPeriodontal Biology LaboratoryUniversidad de Chile Santiago Chile

2. Faculty of DentistryOral Pathology LaboratoryUniversidad Andres Bello Santiago Chile

3. Faculty of DentistryUniversidad Peruana Cayetano Heredia Lima Perú

4. Faculty of SciencesCenter for Integrative BiologyUniversidad Mayor Santiago Chile

5. Faculty of SciencesCenter for Genomic and BioinformaticUniversidad Mayor Santiago Chile

6. Faculty of Health SciencesDentistry UnitUniversidad Autónoma de Chile Santiago Chile

Funder

Fondo Nacional de Desarrollo Científico y Tecnológico

Comisión Nacional de Investigación Científica y Tecnológica

Publisher

Wiley

Subject

Periodontics

Reference57 articles.

1. Inflammatory and immune pathways in the pathogenesis of periodontal disease;Cekici A;Periodontol,2000

2. Host modulation: controlling the inflammation to control the infection;Bartold PM;Periodontol,2000

3. The role of T cells in periodontal disease: homeostasis and autoimmunity;Gemmell E;Periodontol,2000

4. T‐cell phenotype as a risk factor for periodontal disease;Houri‐Haddad Y;Periodontol,2000

5. The oral microbiota: dynamic communities and host interactions

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