Long-term Aβ exposure augments mCa2+-independent mROS-mediated depletion of cardiolipin for the shift of a lethal transient mitochondrial permeability transition to its permanent mode in NARP cybrids: a protective targeting of melatonin-Reference-Cited by-同舟云学术

Long-term Aβ exposure augments mCa2+-independent mROS-mediated depletion of cardiolipin for the shift of a lethal transient mitochondrial permeability transition to its permanent mode in NARP cybrids: a protective targeting of melatonin

Published:2012-08-30 Issue:1 Volume:54 Page:107-125
ISSN:0742-3098
Container-title:Journal of Pineal Research
language:en
Short-container-title:J. Pineal Res.

Author:

Hsiao Chia-Wei¹,Peng Tsung-I.²³,Peng Alexander C.⁴,Reiter Russel J.⁵,Tanaka Masashi⁶,Lai Yiu-Kay¹,Jou Mei-Jie⁷

Affiliation:

1. Department of Life Science and Institute of Biotechnology; National Tsing Hua University; Hsinchu Taiwan

2. Department of Neurology; Kee-Lung Medical Center; Chang Gung Memorial Hospital; Kee-Lung Taiwan

3. Department of Medicine; Chang Gung University; Tao-Yuan Taiwan

4. The Williston Northampton School; Easthampton MA USA

5. Department of Cellular and Structural Biology; Health Science Center; University of Texas; San Antonio TX USA

6. Department of Genomics for Longevity and Health; Tokyo Metropolitan Institute of Gerontology; Tokyo Japan

7. Department of Physiology and Pharmacology; College of Medicine; Chang Gung University; Tao-Yuan Taiwan

Funder

CMRPD

CMRPG

Chang Gung Medical Research Foundation

Publisher

Wiley

Subject

Endocrinology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jpi.12004/fullpdf

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