Prolactin in obese children: a bridge between inflammation and metabolic-endocrine dysfunction

Author:

Chirico Valeria1,Cannavò Salvatore2,Lacquaniti Antonio3,Salpietro Vincenzo1,Mandolfino Mattia2,Romeo Petronilla Daniela2,Cotta Oana2,Munafò Caterina1,Giorgianni Grazia4,Salpietro Carmelo1,Arrigo Teresa1

Affiliation:

1. Department of Pediatric Sciences; University of Messina; Messina; Italy

2. Department of Medicine and Pharmacology; Section of Endocrinology; University of Messina; Messina; Italy

3. Department of Internal Medicine; Section of Nephrology; University of Messina; Messina; Italy

4. Department of Biochemical, Physiological and Nutritional Sciences; Section of Clinical Biochemistry; University of Messina; Messina; Italy

Publisher

Wiley

Subject

Endocrinology, Diabetes and Metabolism,Endocrinology

Reference31 articles.

1. Cause-specific excess deaths associated with underweight, overweight, and obesity;Flegal;JAMA,2007

2. Classical endocrine diseases causing obesity;Weaver;Frontiers of hormone research,2008

3. Adipokines and their role in allergies;Ciprandi;International journal of immunopathology and pharmacology,2011

4. STAT 5 activators can replace the requirement of FBS in the adipogenesis of 3T3-L1 cells;Stewart;Biochemical and Biophysical Research Communications,2004

5. Prolactin upregulates its receptors and inhibits lipolysis and leptin release in male rat adipose tissue;Brandebourg;Biochemical and Biophysical Research Communications,2007

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