Astrocytes release glutamate via cystine/glutamate antiporter upregulated in response to increased oxidative stress related to sporadic amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

Astrocytes release glutamate via cystine/glutamate antiporter upregulated in response to increased oxidative stress related to sporadic amyotrophic lateral sclerosis

Published:2020-12 Issue:6 Volume:40 Page:587-598
ISSN:0919-6544
Container-title:Neuropathology
language:en
Short-container-title:Neuropathology

Author:

Kazama Miku¹,Kato Yoichiro²^ORCID,Kakita Akiyoshi³,Noguchi Noriko⁴,Urano Yasuomi⁴,Masui Kenta²,Niida‐Kawaguchi Motoko²,Yamamoto Tomoko²,Watabe Kazuhiko⁵,Kitagawa Kazuo⁶,Shibata Noriyuki¹

Affiliation:

1. Faculty of Medicine Tokyo Women's Medical University Tokyo Japan

2. Division of Pathological Neuroscience, Department of Pathology Tokyo Women's Medical University Tokyo Japan

3. Department of Pathology Brain Research Institute Niigata University Niigata Japan

4. Department of Medical Life Systems Faculty of Life and Medical Sciences Doshisha University Kyotanabe Japan

5. Department of Medical Technology Kyorin University Tokyo Japan

6. Department of Neurology Tokyo Women's Medical University Tokyo Japan

Publisher

Wiley

Subject

Clinical Neurology,General Medicine,Pathology and Forensic Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/neup.12716

Reference42 articles.

1. Glial Cells—The Strategic Targets in Amyotrophic Lateral Sclerosis Treatment

2. Nitric Oxide-Mediated Oxidative Damage and the Progressive Demise of Motor Neurons in ALS

3. DNA damage accumulates and responses are engaged in human ALS brain and spinal motor neurons and DNA repair is activatable in iPSC-derived motor neurons with SOD1 mutations

4. Identification of a Lipid Peroxidation Product as a Potential Trigger of the p53 Pathway

5. Morphological evidence for lipid peroxidation and protein glycoxidation in spinal cords from sporadic amyotrophic lateral sclerosis patients

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