Endocytosis and toxicity of clostridial binary toxins depend on a clathrin-independent pathway regulated by Rho-GDI-Reference-Cited by-同舟云学术

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Endocytosis and toxicity of clostridial binary toxins depend on a clathrin-independent pathway regulated by Rho-GDI

Published:2010-10-19 Issue:1 Volume:13 Page:154-170
ISSN:1462-5814
Container-title:Cellular Microbiology
language:en
Short-container-title:

Author:

Gibert Maryse,Monier Marie-Noëlle,Ruez Richard,Hale Martha L.,Stiles Bradley G.,Benmerah Alexandre,Johannes Ludger,Lamaze Christophe,Popoff Michel R.

Publisher

Hindawi Limited

Subject

Virology,Immunology,Microbiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1462-5822.2010.01527.x/fullpdf

Reference77 articles.

1. Anthrax toxin triggers endocytosis of its receptor via a lipid raft-mediated clathrin-dependent process;Abrami;J Cell Biol,2003

2. Botulinum C2 toxin ADP-ribosylates actin;Aktories;Nature,1986

3. Interaction of Clostridium botulinum C2 toxin with lipid bilayer membranes and Vero cells: inhibition of channel function in chloroquine and related compounds in vitro and toxin action in vivo;Bachmeyer;FASEB J,2001

4. Cellular uptake of Clostridium botulinum C2 toxin requires oligomerization and acidification;Barth;J Biol Chem,2000

5. Binary bacterial toxins: biochemistry, biology, and applications of common Clostridium and Bacillus proteins;Barth;Microbiol Mol Biol Rev,2004

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