Molecular mechanisms underlying the probiotic effects of Escherichia coli Nissle 1917 involve ZO-2 and PKC? redistribution resulting in tight junction and epithelial barrier repair-Reference-Cited by-同舟云学术

Molecular mechanisms underlying the probiotic effects of Escherichia coli Nissle 1917 involve ZO-2 and PKC? redistribution resulting in tight junction and epithelial barrier repair

Published:2007-03 Issue:3 Volume:9 Page:804-816
ISSN:1462-5814
Container-title:Cellular Microbiology
language:en
Short-container-title:Cell Microbiol

Author:

Zyrek Agata A.,Cichon Christoph,Helms Simone,Enders Corinne,Sonnenborn Ulrich,Schmidt M. Alexander

Publisher

Hindawi Limited

Subject

Virology,Immunology,Microbiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1462-5822.2006.00836.x/fullpdf

Reference64 articles.

1. Protein phosphorylation by protein kinase C in HEp-2 cells infected with enteropathogenic Escherichia coli

2. Intestinal epithelial responses to enteric pathogens: effects on the tight junction barrier, ion transport, and inflammation

3. Bacterial-associated cholera toxin and GM1 binding are required for transcytosis of classical biotype Vibrio cholerae through an in vitro M cell model system

4. Toll-like receptor 2 enhances ZO-1-associated intestinal epithelial barrier integrity via protein kinase C

5. Protein Kinase C Signaling Regulates ZO-1 Translocation and Increased Paracellular Flux of T84 Colonocytes Exposed toClostridium difficile Toxin A

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