Sulforaphane enhances proteasomal and autophagic activities in mice and is a potential therapeutic reagent for Huntington's disease-Reference-Cited by-同舟云学术

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Sulforaphane enhances proteasomal and autophagic activities in mice and is a potential therapeutic reagent for Huntington's disease

Published:2014-01-18 Issue:3 Volume:129 Page:539-547
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Liu Yanying¹,Hettinger Casey L.¹,Zhang Dong¹,Rezvani Khosrow¹,Wang Xuejun¹,Wang Hongmin¹

Affiliation:

1. Division of Basic Biomedical Sciences; Sanford School of Medicine; University of South Dakota; Vermillion South Dakota USA

Funder

NIH

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12647/fullpdf

Reference41 articles.

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2. Apoptosis provoked by the oxidative stress inducer menadione (Vitamin K(3) is mediated by the Fas/Fas ligand system;Caricchio;Clin. Immunol.,1999

3. Mutant huntingtin aggregates impair mitochondrial movement and trafficking in cortical neurons;Chang;Neurobiol. Dis.,2006

4. Crosstalk between Nrf2 and the proteasome: therapeutic potential of Nrf2 inducers in vascular disease and aging;Chapple;Int. J. Biochem. Cell Biol.,2012

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