Protein interacting with NIMA (never in mitosis A)-1 regulates axonal growth cone adhesion and spreading through myristoylated alanine-rich C kinase substrate isomerization-Reference-Cited by-同舟云学术

Protein interacting with NIMA (never in mitosis A)-1 regulates axonal growth cone adhesion and spreading through myristoylated alanine-rich C kinase substrate isomerization

Published:2016-04-08 Issue:5 Volume:137 Page:744-755
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Sosa Lucas J.¹,Malter James S.²,Hu Jie²,Bustos Plonka Florentyna³,Oksdath Mariana³,Nieto Guil Alvaro F.³,Quiroga Santiago³,Pfenninger Karl H.¹

Affiliation:

1. Department of Pediatrics and Colorado Intellectual and Developmental Disabilities Research Center; University of Colorado School of Medicine; Aurora Colorado USA

2. Department of Pathology; University of Texas Southwestern Medical Center; Dallas Texas USA

3. Departamento de Química Biológica-CIQUIBIC; Facultad de Ciencias Químicas; Universidad Nacional de Córdoba-CONICET; Córdoba Argentina

Funder

International Society for Neurochemistry, CAEN Category 1C Return Home

Agencia Nacional de Promoción Científica y Tecnológica

National Institutes of Health

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.13612/fullpdf

Reference59 articles.

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3. A molecular clutch between the actin flow and N-cadherin adhesions drives growth cone migration;Bard;J. Neurosci.,2008

4. Pin1 mediates neural-specific activation of the mitochondrial apoptotic machinery;Becker;Neuron,2006

5. Protein kinase C-stimulated phosphorylation in vitro of a Mr 80,000 protein phosphorylated in response to phorbol esters and growth factors in intact fibroblasts. Distinction from protein kinase C and prominence in brain;Blackshear;J. Biol. Chem.,1986

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