Kallikrein 6 signals through PAR1 and PAR2 to promote neuron injury and exacerbate glutamate neurotoxicity-Reference-Cited by-同舟云学术

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Kallikrein 6 signals through PAR1 and PAR2 to promote neuron injury and exacerbate glutamate neurotoxicity

Published:2013-05-27 Issue:2 Volume:127 Page:283-298
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Yoon Hyesook¹²,Radulovic Maja¹,Wu Jianmin²,Blaber Sachiko I.³,Blaber Michael³,Fehlings Michael G.⁴,Scarisbrick Isobel A.¹²⁵

Affiliation:

1. Neurobiology of Disease Program; Mayo Medical and Graduate School; Rochester Minnesota USA

2. Department of Physical Medicine and Rehabilitation; Mayo Medical and Graduate School; Rochester Minnesota USA

3. Department of Biomedical Sciences; Florida State University College of Medicine; Tallahassee Florida USA

4. Department of Surgery; Toronto Western Research Institute; Toronto Ontario Canada

5. Department of Neurology; Mayo Medical and Graduate School; Rochester Minnesota USA

Funder

The Christopher and Dana Reeve Paralysis Foundation

Collaborative MS Research Award

National Multiple Sclerosis Society

Craig H. Neilsen Foundation

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12293/fullpdf

Reference93 articles.

1. Thrombin and prothrombin are expressed by neurons and glial cells and accumulate in neurofibrillary tangles in Alzheimer disease brain;Arai;J. Neuropathol. Exp. Neurol.,2006

2. Kallikrein-related peptidase 6 in Alzheimer's disease and vascular dementia;Ashby;Brain Res.,2011

3. Basso mouse scale for locomotion detects differences in recovery after spinal cord injury in five common mouse strains;Basso;J. Neurotrauma,2006

4. Bone morphogenetic protein 2 inhibits neurite outgrowth of motor neuron-like NSC-34 cells and up-regulates its type II receptor;Benavente;J. Neurochem.,2012

5. Stress-induced spine loss in the medial amygdala is mediated by tissue-plasminogen activator;Bennur;Neuroscience,2007

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