Tertiary ?Hyperphosphatoninism? Accentuates Hypophosphatemia and Suppresses Calcitriol Levels in Renal Transplant Recipients
Author:
Publisher
Wiley
Subject
Pharmacology (medical),Transplantation,Immunology and Allergy
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-6143.2007.01753.x/fullpdf
Reference41 articles.
1. Post-renal transplantation hypophosphatemia: a review and novel insights
2. Evidence for a PTH-independent humoral mechanism in post-transplant hypophosphatemia and phosphaturia
3. The renal Na+/phosphate cotransporter NaPi-IIa is internalized via the receptor-mediated endocytic route in response to parathyroid hormone
4. Phosphatonins: a new class of phosphate-regulating proteins
5. Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23
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