QT Interval Prolongation in End-Stage Liver Disease Cannot Be Explained by Nonhepatic Factors

Author:

Patel Divyang1,Singh Prabhpreet2,Katz William13,Hughes Christopher14,Chopra Kapil15,Němec Jan13

Affiliation:

1. University of Pittsburgh School of Medicine; Pittsburgh PA

2. Department of Medicine, School of Medicine; University of Pittsburgh Medical Center Pittsburgh; PA

3. Heart and Vascular Institute; University of Pittsburgh Medical Center; Pittsburgh PA

4. Starzl Transplantation Institute; Department of Surgery; University of Pittsburgh Medical Center; Pittsburgh PA

5. Division of Gastroenterology; Hepatology, and Nutrition; University of Pittsburgh Medical Center; Pittsburgh PA

Publisher

Wiley

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,General Medicine

Reference50 articles.

1. [Ventricular tachycardia with 2 variable opposing foci];Dessertenne;Arch Mal Coeur Vaiss,1966

2. Torsade de pointes: The long-short initiating sequence and other clinical features: Observations in 32 patients;Kay;J Am Coll Cardiol,1983

3. Coassembly of K(V)LQT1 and minK (IsK) proteins to form cardiac I(Ks) potassium channel;Sanguinetti;Nature,1996

4. Risk factors for prolonged QTc among US adults: Third National Health and Nutrition Examination survey;Benoit;Eur J Cardiovasc Prev Rehabil,2005

5. [Ventricular tachycardia “with reversal of points” and electrolyte disorders];Klauser;Schweiz Med Wochenschr,1977

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