α-Lecithin: Cholesterol Acyltransferase Deficiency
Author:
Publisher
Wiley
Subject
Internal Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.0954-6820.1987.tb09923.x/fullpdf
Reference12 articles.
1. Fish-eye disease. A new familial condition with massive corneal opacities and dyslipoproteinemia;Carlson;Lancet,1979
2. Evidence for deficiency of high density lipoprotein lecithin: cholesterol acyltransferase activity (α-LCAT) in fish eye disease;Carlson;Acta Med Scand,1985
3. Human plasma lecithin-cholesterol acyltransferase;Aron;J Biol Chem,1978
4. Fish eye disease: a new familial condition with massive corneal opacities and dyslipo-proteinaemia;Carlson;Eur J Clin Invest,1982
5. Determination of serum triglycerides;Carlson;J Atheroscler Res,1963
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1. Substrate Specificity of Plasma Lecithin: Cholesterol Acyltransferase in Abetalipoproteinemia;Acta Medica Scandinavica;2009-04-24
2. Structural Differences Between Wild-type and Fish Eye Disease Mutant of Lecithin:cholesterol Acyltransferase;Journal of Biomolecular Structure and Dynamics;2006-08
3. Deletion of N-terminal amino acids from human lecithin:cholesterol acyltransferase differentially affects enzyme activity toward α- and β-substrate lipoproteins;Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics;2003-03
4. Relationship between structure and biochemical phenotype of lecithin:cholesterol acyltransferase (LCAT) mutants causing fish-eye disease;Journal of Lipid Research;2000-05
5. Phospholipase A2: Its Usefulness in Laboratory Diagnostics;Critical Reviews in Clinical Laboratory Sciences;1999-01
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