Src regulates insulin secretion and glucose metabolism by influencing subcellular localization of glucokinase in pancreatic β‐cells-Reference-Cited by-同舟云学术

Src regulates insulin secretion and glucose metabolism by influencing subcellular localization of glucokinase in pancreatic β‐cells

Published:2015-09-13 Issue:2 Volume:7 Page:171-178
ISSN:2040-1116
Container-title:Journal of Diabetes Investigation
language:en
Short-container-title:J Diabetes Invest

Author:

Sato Hiroki¹,Nagashima Kazuaki¹,Ogura Masahito¹,Sato Yuichi¹,Tahara Yumiko¹,Ogura Kasane¹,Yamano Gen¹,Sugizaki Kazu¹,Fujita Naotaka¹,Tatsuoka Hisato¹,Usui Ryota¹,Mukai Eri²,Fujimoto Shimpei³,Inagaki Nobuya¹

Affiliation:

1. Department of Diabetes, Endocrinology and Nutrition Graduate School of Medicine Kyoto University Kyoto Japan

2. Department of Medical Physiology Graduate School of Medicine, Chiba University Chiba Japan

3. Department of Endocrinology, Metabolism, and Nephrology Kochi Medical School Kochi University Nankoku Japan

Funder

Ministry of Agriculture, Forestry and Fisheries

Core Research for Evolutional Science and Technology, Japan Science and Technology Agency

Publisher

Wiley

Subject

General Medicine,Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jdi.12407

Reference43 articles.

1. Mitochondrial function in normal and diabetic β-cells

2. Stress and the inflammatory process: a major cause of pancreatic cell death in type 2 diabetes;Montane J;Diabetes Metab Syndr Obes,2014

3. Role of endogenous ROS production in impaired metabolism-secretion coupling of diabetic pancreatic β cells

4. Src activation generates reactive oxygen species and impairs metabolism–secretion coupling in diabetic Goto–Kakizaki and ouabain-treated rat pancreatic islets

5. Exendin-4 Suppresses Src Activation and Reactive Oxygen Species Production in Diabetic Goto-Kakizaki Rat Islets in an Epac-Dependent Manner

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