Epithelial miR‐215 negatively modulates Th17‐dominant inflammation by inhibiting CXCL12 production in the small intestine-Reference-Cited by-同舟云学术

Epithelial miR‐215 negatively modulates Th17‐dominant inflammation by inhibiting CXCL12 production in the small intestine

Published:2022-02-06 Issue:4 Volume:27 Page:243-253
ISSN:1356-9597
Container-title:Genes to Cells
language:en
Short-container-title:Genes to Cells

Author:

Pareek Siddhika¹,Sanchenkova Xenia²,Sakaguchi Taiki²³,Murakami Mari²³,Okumura Ryu²³,Kayama Hisako²³⁴,Kawauchi Saya²³,Motooka Daisuke²⁵,Nakamura Shota²⁵,Okuzaki Daisuke²⁵,Kishimoto Tadamitsu²,Takeda Kiyoshi²³^ORCID

Affiliation:

1. Regenerative Medicine Institute Cedars‐Sinai Medical Center Los Angeles California USA

2. WPI Immunology Frontier Research Center Osaka University Osaka Japan

3. Laboratory of Immune Regulation Department of Microbiology and Immunology Graduate School of Medicine Osaka University Osaka Japan

4. Institute for Advanced Co‐Creation Studies Osaka University Osaka Japan

5. Genome Information Research Center Research Institute for Microbial Diseases Osaka University Osaka Japan

Funder

Japan Agency for Medical Research and Development

Publisher

Wiley

Subject

Cell Biology,Genetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/gtc.12922

Reference36 articles.

1. MicroRNAs

2. p53-Responsive MicroRNAs 192 and 215 Are Capable of Inducing Cell Cycle Arrest

3. Control of cell proliferation pathways by microRNAs

4. CXCR4 inhibitor attenuates ovalbumin-induced airway inflammation and hyperresponsiveness by inhibiting Th17 and Tc17 cell immune response

5. CXCL12 Is a constitutive and inflammatory chemokine in the intestinal immune system

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