Blood group B gene is barely expressed inin vitroerythroid culture of Bm-derived CD34+cells without an erythroid cell-specific regulatory element

Author:

Sano R.1,Nogawa M.2,Nakajima T.1,Takahashi Y.1,Takahashi K.1,Kubo R.1,Kominato Y.1,Yokohama A.3,Tsukada J.4,Yamao H.5,Kishida T.6,Ogasawara K.2,Uchikawa M.7

Affiliation:

1. Legal Medicine; Gunma University Graduate School of Medicine; Maebashi Japan

2. Division of Blood Research; Japanese Red Cross Central Blood Institute; Tokyo Japan

3. Blood Transfusion Service; Gunma University Hospital; Maebashi Japan

4. Cancer Chemotherapy Center and Hematology; University of Occupational and Environmental Health; Kitakyusyu Japan

5. Obstetrics and Gynecology; Inoue Hospital; Nobeoka Japan

6. Legal Medicine; Oita University School of Medicine; Yuhu Japan

7. Division of Blood Research; Japanese Red Cross Kanto-Koshinetsu Block Blood Center; Tokyo Japan

Publisher

Wiley

Subject

Hematology,General Medicine

Reference35 articles.

1. Human Blood Groups

2. Molecular genetics of ABO;Yamamoto;Vox Sang,2000

3. Expression of ABO blood-group genes is dependent upon an erythroid cell-specific regulatory element, which is deleted in persons with the Bm phenotype;Sano;Blood,2012

4. Mutation of the GATA site in the erythroid cell-specific regulatory element of the ABO gene in a Bm subgroup individual;Nakajima;Transfusion,2013

5. Deletion of the RUNX1 binding site in the erythroid cell-specific regulatory element of the ABO gene in two individuals with the Am phenotype;Takahashi;Vox Sang,2014

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