Amelioration of lesions associated with 24-hour suboptimal platelet storage at 16 °C by a p38MAPK inhibitor, VX-702
Author:
Affiliation:
1. Blood Components Department; American Red Cross Holland Laboratory; Rockville MD USA
Publisher
Wiley
Subject
Hematology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/vox.12221/fullpdf
Reference18 articles.
1. Overview on platelet preservation: better controls over storage lesion;Ohto;Transfus Apher Sci,2011
2. Tumor necrosis factor-α-converting enzyme (ADAM17) mediates GPIbα shedding from platelets in vitro and in vivo;Bergermeier;Circ Res,2004
3. p38 mitogen-activated protein kinase activation during platelet storage: consequences for platelet recovery and hemostatic function in vivo;Canult;Blood,2010
4. Platelet storage lesion: analysis of molecular mechanisms by targeting protein kinases;Schubert;Transfusion,2010
5. An inhibition of p38 mitogen activated protein kinase delays the platelet storage lesion;Skripchenko;PLoS ONE,2013
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1. p38MAPK Signaling Pathway in Osteoarthritis: Pathological and Therapeutic Aspects;Journal of Inflammation Research;2022-02
2. P38 mitogen activated protein kinase inhibitor improves platelet in vitro parameters and in vivo survival in a SCID mouse model of transfusion for platelets stored at cold or temperature cycled conditions for 14 days;PLOS ONE;2021-05-11
3. Targeting Intraplatelet Signaling Pathways as Potential Antithrombotic Strategy;Platelets in Thrombotic and Non-Thrombotic Disorders;2017
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