Pridopidine modifies disease phenotype in a SOD1 mouse model of amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

Pridopidine modifies disease phenotype in a SOD1 mouse model of amyotrophic lateral sclerosis

Published:2022-02-12 Issue:5 Volume:55 Page:1356-1372
ISSN:0953-816X
Container-title:European Journal of Neuroscience
language:en
Short-container-title:Eur J of Neuroscience

Author:

Estévez‐Silva Héctor M.¹²^ORCID,Mediavilla Tomás¹^ORCID,Giacobbo Bruno Lima¹^ORCID,Liu Xijia³^ORCID,Sultan Fahad R.¹^ORCID,Marcellino Daniel J.¹^ORCID

Affiliation:

1. Department of Integrative Medical Biology Umeå University Umeå Sweden

2. Departamento de Ciencias Médicas Básicas, Instituto de Tecnologías Biomédicas (ITB) Universidad de La Laguna Santa Cruz de Tenerife Spain

3. Umeå School of Business, Economics and Statistics Umeå University Umeå Sweden

Funder

Kempestiftelserna

Agencia Canaria de Investigación, Innovación y Sociedad de la Información

Publisher

Wiley

Subject

General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/ejn.15608

Reference59 articles.

1. A comprehensive assessment of the SOD1G93A low-copy transgenic mouse, which models human amyotrophic lateral sclerosis

2. Effect of transgene copy number on survival in the G93A SOD1 transgenic mouse model of ALS

3. A mutation in sigma-1 receptor causes juvenile amyotrophic lateral sclerosis

4. Increased TCA cycle intermediates in response to diet with Deanna protocol in ALS mouse model (578.3)

5. Fitting Linear Mixed-Effects Models Usinglme4

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