SLPI in periodontal Ligament is not sleepy during biophysical force‐induced tooth movement
Author:
Affiliation:
1. Dental Science Research Institute School of Dentistry Chonnam National University Gwangju Korea
2. Department of Anatomy and Neuroscience College of Medicine Eulji University Daejeon Korea
Funder
National Research Foundation of Korea
Publisher
Wiley
Subject
Periodontics
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcpe.13416
Reference36 articles.
1. Inflammation and Tooth Movement: The Role of Cytokines, Chemokines, and Growth Factors
2. IL-8 and MCP-1 induced by excessive orthodontic force mediates odontoclastogenesis in periodontal tissues
3. TNF-α and IL-1β inhibit RUNX2 and collagen expression but increase alkaline phosphatase activity and mineralization in human mesenchymal stem cells
4. Inflammatory Cytokines Induce a Unique Mineralizing Phenotype in Mesenchymal Stem Cells Derived from Human Bone Marrow
5. Secretory Leukocyte Protease Inhibitor Reverses Inhibition by CNS Myelin, Promotes Regeneration in the Optic Nerve, and Suppresses Expression of the Transforming Growth Factor- Signaling Protein Smad2
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3. Effect of local application of bone morphogenetic protein -2 on experimental tooth movement and biological remodeling in rats;Frontiers in Physiology;2023-04-18
4. Sclerostin in periodontal ligament: Homeostatic regulator in biophysical force‐induced tooth movement;Journal of Clinical Periodontology;2022-07-04
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