Differential cytotoxicity, ER/oxidative stress, dysregulated AMPKα signaling, and mitochondrial stress by ethanol and its metabolites in human pancreatic acinar cells-Reference-Cited by-同舟云学术

Differential cytotoxicity, ER/oxidative stress, dysregulated AMPKα signaling, and mitochondrial stress by ethanol and its metabolites in human pancreatic acinar cells

Published:2021-04-02 Issue:5 Volume:45 Page:961-978
ISSN:0145-6008
Container-title:Alcoholism: Clinical and Experimental Research
language:en
Short-container-title:Alcohol Clin Exp Res

Author:

Srinivasan Mukund P.¹^ORCID,Bhopale Kamlesh K.¹,Caracheo Anna A.¹,Kaphalia Lata²^ORCID,Loganathan Gopalakrishnan³,Balamurugan Appakalai N.³⁴,Rastellini Cristiana²⁵⁶,Kaphalia Bhupendra S.¹^ORCID

Affiliation:

1. Department of Pathology The University of Texas Medical Branch Galveston TX USA

2. Department of Internal Medicine The University of Texas Medical Branch Galveston TX USA

3. Department of Surgery University of Louisville Louisville KY USA

4. Islet Biology Laboratory Division of Pediatric General and Thoracic Surgery Cincinnati Children's Hospital Medical Center Department of Surgery University of Cincinnati Cincinnati OH USA

5. Department of Neuroscience & Cell Biology The University of Texas Medical Branch Galveston TX USA

6. Department of Microbiology & Immunology The University of Texas Medical Branch Galveston TX USA

Funder

National Institute on Alcohol Abuse and Alcoholism

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acer.14595

Reference76 articles.

1. Hepatic alcohol dehydrogenase deficiency induces pancreatic injury in chronic ethanol feeding model of deer mice