Role of MyD88-dependent and MyD88-independent signaling inPorphyromonas gingivalis-elicited macrophage foam cell formation
Author:
Affiliation:
1. Section of Infectious Diseases; Department of Medicine; Boston University Medical Center; Boston; MA; USA
2. Cold Spring Harbor Laboratory; Cold Spring Harbor; NY; USA
Publisher
Wiley
Subject
Microbiology (medical),General Dentistry,Immunology,Microbiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/omi.12003/fullpdf
Reference73 articles.
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2. Porphyromonas gingivalis lipopolysaccharide displays functionally diverse interactions with the innate host defense system;Bainbridge;Ann Periodontol,2002
3. The role of immune responses in bone loss during periodontal disease;Baker;Microbes Infect,2000
4. CD4+ T cells and the proinflammatory cytokines gamma interferon and interleukin-6 contribute to alveolar bone loss in mice;Baker;Infect Immun,1999
5. The periodontal pathogen Porphyromonas gingivalis cleaves apoB-100 and increases the expression of apoM in LDL in whole blood leading to cell proliferation;Bengtsson;J Intern Med,2008
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